Fundus diseases of optic nerve atrophy _ Beijing Chaoyang-Western

Optic atrophy (opticatrophy) medicine called green blind, refers to the eye, eye appearance-God no barrier coating look sky, but considers himself not as the main symptoms of cataract eye disease. Basic pathogenesis as head of sweat pore obstruction due to Visual, unknown. Its formation and hepatic and renal losses, essence and blood shortage asdthenic, fine not; prolonged blood loss; fever late, Yin-loss injury; seven emotions stagnation, liver failure, da. In addition, trauma, repression, postoperative, the head of damage, choroid stasis, sweat pore close resistance can also be formed in the disease, diseases of a deficiency of Lord. Optic atrophy is not a disease, and refers to any disease-induced retinal ganglion cells and its axonal lesion occurs, all changes of the optic nerve of a form of change, as a common noun pathology, typically occurs in the retina to the lateral geniculate nucleus of ganglion cells Axon degeneration. Optic atrophy is the end result of optic nerve lesions. Performance for the optic nerve of degeneration and disappear, conduction disorder, vision changes, beauty and lost. Generally divided into two categories of primary and secondary, in addition to the above symptoms, fundus examination remains visible as the nipple color is light yellow or pale color, blur, physiological depression disappear, blood vessels become thin, etc. Modern Western medicine on the disease still lack specific therapy. There are many reasons which cause can cause, common with ischemia, inflammation, oppression, trauma and demyelinating diseases are as follows: 1. due to intracranial hypertension secondary to optic atrophy 2. intracranial inflammation, prevalent tuberculous meningitis or optic CHIASM arachnoiditis 3. retinopathy ⑴ vascular, central retinal artery or vein occlusion, optic nerve itself of atherosclerosis, vascular disorder, normal nutrition bleeding (digestive and uterus, etc.) ⑵ inflammatory glaucoma after ⑶ ⑷ retinitis pigmentosa ⑸ s u R e f m disease ⑹ black Mont familial dementia 4. optic neuritis and optic nerve lesions, such as the ⑴ vascular ischemic optic neuropathy ⑵ demyelinating disease lack ⑶ vitamin ⑷ because of lead or other metal such as poisoning ⑸ Herpes Zoster ⑹ syphilis sex 5. oppressive caused by tumors, including meningiomas, craniopharyngiomas, pituitary adenoma, aneurysm (anterior communicating artery aneurysm) bone disease, Paget Disease, malformations including Osteitis, skull stricture disease orbital tumors 6. trauma 7. metabolic diseases such as diabetes, gangliosides disease 8. hereditary disease Leber disease, cerebellar ataxia, peripheral neuropathy as Chareot-Marie-Tooth disease 9. nutritional optic atrophy 10. Miscellaneous in children due to more complex are as follows: 1. chromosomal abnormality cat syndrome, partial deletion of the long arm of chromosome 18. lipid disease Tay-sachs disease, Sandhoffs disease, lactose acyl (nerve) of sphingosine poisoning, NIEMANN-Pieck disease, α-β-lipoprotein levels (Bassen-kornzwig syndrome), 3. the variegates disease Hurlers Mucopolysaccharide Diseases, disease with urinary cystine 4. mineral metabolism defects and metabolism Menkes disease, juvenile diabetes, pancreatic cystic fibrosis degeneration, systemic ganglion Glycoside lipid disease, Alzheimer's disease, Albers-Sch Zellwage & ouml; nberg disease 5. hereditary retinal dystrophy Kesrns-Sayer Ushers syndrome, syndrome, Alstrom syndrome 6. gray matter with Battens disease, infantile neuroaxonal dystrophy, Hallervorden-Spatz disease 7. cerebellar ataxia Behr optic atrophy, Marie ataxia, hereditary motor and sensory Polyneuropathy, Charcot-Marie-Tooth disease, olive-Pontine cerebellar degeneration of the white matter lesions 8. primary abnormality of the brain white matter disease, degeneration, Krabbers sponge quality white matter degeneration (Canaran), anti-wrinkles Sudan white matter disease, degeneration, Merzbacher-pelizaeus Cockayne syndrome 9. demyelinating diseases of the adrenal gland white matter degeneration, multiple sclerosis 10. familial optic atrophy, Leber optic atrophy of infantile (implicit and explicit) 11. intracranial fake brain tumor, intracranial hemorrhage, skull stenosis, aqueduct blocking Hydrocephalus manifestation mainly for beauty and disc is grey or pale. Normal disc tone is a variety of factors. Normally, the disc is a temporal color most the nose side is light, and the degree of temporal dimmed and the size of the physiological Cup. Baby video disc color often fade, or check the oppression caused ischemic optic disc of the eyeball. Therefore not solely on the basis of a video disc structure and color correctly diagnosis of optic atrophy, must observe the retinal vascular and peripapillary nerve fiber layer has not changed, particularly in the field of vision, color vision checkedEnquiries, comprehensive analysis, in order to clarify the extent of the optic disc color pale. Peripapillary nerve fiber layer defect may appear fissured or wedge-shaped defects, which turned into a black, for retinal pigment layer exposed; the latter is a red, choroidal exposure. If damage occurs to the upper and lower edge of the area of the disc, then easier to identify because the area nerve fiber layer special thickening, if lesions from disc, since these regional nerve fiber Guide becomes thin, hard to find. Peripapillary accompanied by focal atrophy often prompt nerve fiber layer has the disease, nerve fiber layer in thinning. Although commonly used ophthalmoscopy can be found, but with no red light ophthalmoscope and fundus photography easier to check. Optic disc small vessel usually 9 ~ 10 root, optic atrophy, these will reduce the number of small blood vessels. With fashion shows retinal artery become thin and narrow, occlusion, but the phenomenon is not there all of optic atrophy, usually after the ball had no effect of optic nerve atrophy, if optic atrophy associated with retinal vascular changes, you must directly affect the retinal vascular retinal vessels can occur. Commonly used optic atrophy is divided into two primary and secondary: former disc realm, physiological depression and screen visible; the latter realm fuzzy, physiological depression and sieve is not visible. Vision inspection should note applies small red depending on the subject, visible Center of dark, nasal defect, temporal Island view, concentric shrinking horizon to tubular double blind a temporal perspective too. Color vision disorders often acquired acquired, red green barrier between, hue arrange check inspection of lex specialis derogat legi generali. Fluorescein angiography early meaning, advanced visible disc fluorescence weakened and late strong fluorescence. Vision electrophysiology testing including electroretinography (ERG), ocular figure (EOG) and Visual evoked potentials (VEP) to diagnose illness and prognosis are a secondary meaning. Diagnosis of optic disc based only on the degree of the eyes or pale gray is not confirmed, you must use the Visual function check to diagnosis. Because the disease can have a variety of reasons, it must be possible at the same time make the diagnosis. First of all should exclude intracranial space-occupying lesions, complemented by skull x-ray and other checks, usually included in the General, other cranial CT and MRI also selective application. Treatment measures etiological treatment for first, second, to provide comprehensive treatment. Once the optic atrophy, especially in the course of a long, to make it almost impossible to recover, but the remaining nerve fiber recovery or maintenance of its functionality is entirely possible. Therefore should enable patients with confidence and adherence to treatment. Application of various drugs failed to adopt a strict double-blind test (sometimes not allowed), it is difficult to describe what kind of drugs and methods must be valid. Drugs commonly used include Neurotrophic drugs such as vitamin B1, B12, ATP and Coenzyme A, Vasodilator Drugs and stasis drug such as tobacco, acid, bendazol, vitamin E, venoruton, Salvia, etc. Recently by HBO, counterpulsation injection 654-2 have achieved certain results. There should be mentioned that the Beijing Chaoyang hospital treatment in the use of traditional Chinese medicine has long proved effective, stasis eyesight capsule with Pearl red eyesight eye drops in clinical application on almost 20 years, their blood stasis, diuresis, dredging effects can improve the metabolism of retinal circulation, promotion, significant effect, may continue to be widely used. Small hint: online too long carefully marked impairment of optic nerve atrophy vision to see things not clear or Outlook bridging, Mydriasis, afraid of the light, if the above symptoms, to get attention. In the youth population, due to improper eye, optic neuritis occurs with increasing tendency. This kind of optic nerve atrophy, will lead to blindness. Beijing-Chaoyang Hospital of expert advise, eye pain, or accompanied by headache, vomiting, patients need to hurry up. Also may not have any symptoms, and this requires regular eye checks, especially the genetic history of, or the recent apparent decline in Visual acuity, field of vision narrows.